Early Results Boost Hopes for Historic Gene Editing Attempt

By Associated Press – Re-Blogged From Newsmax

Early, partial results from a historic gene editing study give encouraging signs that the treatment may be safe and having at least some of its hoped-for effect, but it’s too soon to know whether it ultimately will succeed.

The results announced Wednesday are from the first human test of gene editing in the body, an attempt to permanently change someone’s DNA to cure a disease — in this case, a genetic disorder called Hunter syndrome that often kills people in their teens.

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Promising New Alzheimer’s Research Suggests Dementia Can Be Reversed

Re-Blogged From Newsmax Health

After decades of disappointing findings in new Alzheimer’s disease research, scientists have hit on what many experts believe is a promising new way to reverse the condition that could be a game changer in treating the memory-robbing disorder.

A new Cleveland Clinic study found that amyloid protein plaques that accumulate in the brain, which are hallmarks of Alzheimer’s, were eliminated when a researchers removed a naturally occurring enzyme — called BACE1 — from the brains of mice genetically engineered to have a rodent form of the disease.

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New Discoveries in Fight Against Antibiotic Resistance

By – Re-Blogged From  www.seeker.com

Key insights into how bacteria become resistant to antibiotics could reverse the process and renew the efficacy of commonly prescribed medications.

New Research Suggests Memory Loss in Alzheimer’s Patients May be Reversible

By Karla Lant – Re-Blogged From Futurism

New research from a team at MIT indicates symptoms of Alzheimer’s disease (AD) affecting patient’s memories may be reversible. AD causes memory loss by setting up genetic “blockades” formed when the enzyme HCAC2 condenses the genes of the brain responsible for memory. Eventually, those genes become useless; unexpressed, the genes are unable to cause the formation of new memories or retrieval of existing ones.

Clearly, blocking HCAC2 in the brain is an obvious fix; however, it has to date been impossible, in that all prior attempts have negatively affected the internal organs which require other enzymes in the histone deacetylase (HDAC) family for normal function. Researchers at MIT have now found something they hope might be the answer: LED lights which they use to prevent HCAC2 alone from binding with Sp3, its genetic blockade formation partner in crime (and Alzheimer’s).

Image Credit: jarmoluk/Pixabay

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